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Description

10.1073/pnas.2309317120 Proc

Succinate predisposes mice to atrial fibrillation by impairing mitochondrial function via SUCNR1/AMPK axis

It catalyzes the production of superoxide and hydrogen peroxide, which increase lipid peroxidation, iron accumulation, and mitochondrial damage, thus driving ferroptosis (77)

This cellular and molecular damage accelerates the aging process, driving the onset of age-related diseases such as cardiovascular disorders, neurodegenerative diseases, diabetes, and inflammatory conditions