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Craniofacial and skeletal abnormalities in RAR double mutants

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In contrast, for those diseases caused by NRF2 loss of function or a defect in one of the NRF2-regulated pathways, reactivating an alternative pathway that could also contribute to ROS detoxification might be a way to resolve the problem

Branched-chain amino acid catabolism promotes thrombosis risk by enhancing Tropomodulin-3 Propionylation in platelets

Statistics show that globally, PE accounts for at least 46,000 maternal and 500,000 perinatal deaths annually, with its incidence and mortality rates increasing each year 2