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In gastric cancer, SHMT2 exerts a non-metabolic role by interacting with hypoxia-inducible factor 1 (HIF1) and inhibiting its ubiquitination, thereby stabilizing HIF1 protein under hypoxic conditions

Its best-supported mechanism in animal work is a rapid rise in BDNF and its receptor TrkB in the hippocampus, the memory structure, which is a far more concrete claim than neurotrophic. It is established enough in its home country to sit on Russias official list of vital and essential medicines, where it was added in 2011

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doi: 10.1038/ng.257, PMID: [DOI] [PMC free article] [PubMed] [Google Scholar] 46.Kozlitina J, Smagris E, Stender S, Nordestgaard BG, Zhou HH, Tybjaerg-Hansen A, et al

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