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"Recommendations for the identification of chronic hepatitis C virus infection among persons born during 1945-1965" (PDF)

Nonetheless, it is apparent that selenium deficiency, dysregulation of selenoproteins, and genetic variations in selenoprotein genes serve as potential risk factors for OA

doi: 10.3389/fvets.2025.1512701, 4 ChenY.OuZ.PangM.TaoZ.ZhengX.HuangZ.et al

Evidence from other neurodegenerative diseases suggests that the accumulation of mtDNA mutations itself may act as a trigger of microglial activation (95), supporting the possibility that metabolic changes in glia may alter glial inflammatory responses