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The extensive physiological actions of omega-3 PUFAs involve neurons, microglia, and astrocytes, facilitating the modulation of neuroinflammation, pain signal propagation, mitochondrial dysfunction, oxidative stress, and mood dysregulation, all of which are integral to the complex pathology of migraines (80, 81)

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Noncanonical WNT-5A signaling impairs endogenous lung repair in COPD

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This oxidative damage severely impairs the mitochondria's ability to produce adenosine triphosphate (ATP), the currency of cellular energy