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Benoit, Environ

In the screen against the glutathione-deficient yeast strain (Screen I), where any compound inhibiting ChaC1 would permit the growth of these cells (greater than three times the standard deviation of the mean library effect), were considered for further evaluation (Table S2)

genetically engineered mouse models (GEMMs) simulate tumor development in an immune-competent context with pertinent genetics, although they are time-intensive and expensive

Endogenous ROS levels are increased in replicative senescence in human bone marrow mesenchymal stromal cells

A small proportion of ACSL4-positive cells were positive for F4/80, suggesting that some Kupffer cells also underwent ferroptosis