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Although some feel that MxA may be a primary event, classic studies have shown that this is a secondary innate immunity molecule and in distinct contrast to the primary process of complement-mediated microvasculopathy, as highlighted above, which: a) precedes all pathologic events, b) explains the noted ischemia and perifascicular atrophy, and c) explains the impressive improvement after complement inhibition by IVIg based on two controlled trials (34)

Their robust performance in challenging environments ensures that operations are safe and compliant