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Furthermore, OHP causes damage to both neuronal and nonneuronal mitochondria, leading to oxidative stress burden mediated by the redox-sensitive TRPA1 channels, which correlate with OHP-induced cold and mechanical hypersensitivity [71, 115,116,117]

Basingstoke, England: Macmillan Higher Education

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Furthermore, nanodevices can be devised for microenvironment-specific treatment, such as acidosis, oxidative stress, and enzyme-specific release, ensuring controlled release of the drug in the site of injury