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269 High-mobility group box protein 1 (HMGB1)/Toll-like receptor (TLR) and NF-B signaling pathways in neuroinflammation Various immune cells, as well as the corresponding cellular products, are associated with oxidative stress and necrosis activate the innate immune system, probably via the TLR signaling pathway

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Collectively, these findings underscore the regulatory role of PSPH, facilitating the increased expression of galectin-1, offering therapeutic opportunities to overcome phenotypically immunosuppressive GBMs, and augment the efficacy of immunotherapies

In cellular stress induced by MPP+, the knockdown of ATF-4 or CTH resulted in a reduction of GSH levels [43, 44]

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