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In summary, monocyte-derived macrophages from individuals with CD have increased susceptibility to H 2 O 2 -induced ferroptosis cell death, that may be facilitated, at least in part, by reduced expression of the antioxidant GPx1

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It is reported that BSO inhibits the synthesis of the -glutamylcysteinesynthetase enzyme, which leads to the depletion of glutathione (GSH), thus negatively modulates the cellular redox potential

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