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NK cells exhibit strong metabolic flexibility upon short-term cytokine stimulation, but prolonged activation leads to increased mitochondrial metabolism and glycolysis involving mammalian target of rapamycin complex 1 (mTORC1) ( Metabolomic and lipidomic studies have identified distinct alterations in glycerophospholipids among different NK cell subsets ( Figure 3 )

Simultaneous detection and quantification of mitochondrial DNA deletion(s), depletion, and over-replication in patients with mitochondrial disease

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Evidence from a systematic review and meta-analysis

Precisely add 1.0 mL of 0.1% formic acid aqueous solution to the residue, sonicate for 1 min to dissolve the residue, and filter through a 0.22 m membrane for analysis by liquid chromatography-tandem mass spectrometry