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For instance, when Glu accumulates at synapses, the Arg- and hArg-generated NO aggravate the Glu excitotoxicity, accelerating neurodegeneration ( + /K + -ATPase activity are among contributors to the brain damage by Arg-derived guanidino compounds ( Based on the data considered above ( metabolism and signaling, which contribute to the SCI-induced inflammation in cerebral cortex, may be manifested in the levels of the amino acids and GSH involved (Figure 1)

Because Globular Actin is abundant in nearly all cell types, Thymosin Beta-4 influences any cell requiring structural reorganization or migration (e.g., a skin cell moving to close a wound, or a progenitor cell moving to damaged heart muscle) [6]

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Journal of Physiology (Paris), 91(3), 173178