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Furthermore, oxidative stress and inflammatory pathways triggered by mitochondrial dysfunction also have a crucial impact on aging

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The integrated analysis of the experimental X-ray crystallography data herein presented with computational analysis and literature data on homologous enzymes strongly supports the identification of at least two druggable sites on SmTGR, i.e., sites S3 (doorstop) and S8, the later described for the first time here

Our model simulations suggest that chronic usage of APAP at recommended therapeutic levels probably does mild liver damage and may be associated with a reduction in GSH levels that compromise antioxidant defense capacity

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