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In the cellular context, these aberrant interactions can disrupt physiological processes by sequestering various proteins essential for normal cellular functions, resulting in diffused aggregates that may act as molecular triggers for LB nucleation

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Reactive oxygen species (ROS) or free radicals cause damage to key cellular components like DNA, RNA, lipids, proteins, and structural membranes

Atovaquone, a repurposed antimalarial drug, acts as a selective OXPHOS inhibitor by targeting the CoQ10-dependent mitochondrial complex III, causing electron leakage, ROS production, and cell death, as observed in Fhit-deficient lung cancer cell lines