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This inhibition attenuates the mesenchymal phenotype of BCSCs, suppresses their self-renewal capabilities, and enhances sensitivity to apoptosis, making BYL-719 a potent therapeutic agent in targeting EMT-related resistance in breast cancer [34]

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Similarly, the concomitant downregulation of Adam19, a metalloproteinase closely associated with inflammation and oxidative stress-induced tissue remodeling 56 , further reinforces the hypothesis that APP treatment actively promotes endogenous antioxidant defenses and improves redox homeostasis rather than provoking nonspecific oxidative damage