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This cellular defense mechanism against ferroptosis operates independently of GPX4 and FSP1 (Fig

10.2174/1381612825666190903155321 106 RathnamS

The MALDI-imaging results were consistent with those of the MALDITOFMS analysis and suggested that allicin altered lipid metabolism in the cerebral infarct area, especially arachidonic acid metabolism, which may be involved in post-stroke functional impairment