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In a strong correlation with its inhibitory effect on LPS-induced I-B kinase (IKK) activation, I-B degradation, nuclear translocation of NF-B, and binding of NF-B to the B motif of the iNOS promoter, lutein suppressed LPS-induced NF-B activation

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sedebokerense , which made it possible to block the infection by using inhibitors targeting CAZymes or methionine biosynthesis [39, 40]

The other regulation process can operate at the protein level, as was demonstrated in a study of Z

Am J Clin Nutr 2003