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Despite the above challenges, recent publications suggest that the application of omics technology to the clinical setting of acute APAP toxicity can generate candidate biomarkers for further examination in future studies conducted with more optimized study designs

Our identification of VRK2-mediated SLC7A11 trafficking suggests a potential immune evasion mechanism, as SLC7A11-high tumors often display T cell exclusion phenotypes [37], warranting future investigation into whether VRK2 expression could predict therapeutic response to combined ferroptosis inducers and immune checkpoint inhibitors

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2024 Jul 15];24(7):1401-13

Scavenging reactive oxygen species selectively inhibits M2 macrophage polarization and their pro-tumorigenic function in part, via Stat3 suppression