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Results revealed that pre-treatment with either EBV or LMP1 significantly enhanced mitochondrial replication (see Figure 4A), intracellular ATP levels (see Figure 4B), and mitochondrial membrane potential (see Figure 4C

Moreover, IL1B/IL-1 (interleukin 1 beta) stimulation leads to KAT2B (lysine acetyltransferase 2B)-dependent acetylation and activation of NNT (nicotinamide nucleotide transhydrogenase) in gastric cancer cells, resulting in increased production of NADPH and the preservation of Fe-S clusters within mitochondria [189] (Fig

Indeed, our Compass analysis reflected a global increase in glucose catabolism in FoB when Gclc was ablated (Fig

Schliebs R, et al

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