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This applies to glucose-metabolizing enzymes (e.g., hexokinase, aldolase, lactate dehydrogenase, phosphofructokinase, and PDH), which increase considerably in activity between the ages of 15 and 30 days (Takagaki, 1974

c-Met represents a potential therapeutic target for personalized treatment in hepatocellular carcinoma

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Songs research on non-small cell lung cancer (NSCLC) found that there is ferroptosis in cancer cells treated with cisplatin, and ferroptosis-resistant cancer cells could secrete miR-4443 through exosomes, which increase the expression of FSP1 in ferroptosis-sensitive cancer cells, thereby enhancing the resistance of the cancer cells to ferroptosis [29]