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This, in turn, would affect ion-motive ATPases (6, 21, 131, 135, 138141), which would cause diminution of the neuronal cell potential with corresponding loss of the ability of the neuron to participate in neurotransmission, which would be expected in a disease associated with loss of cognition

References 1 AkyuzE.DoganyigitZ.ErogluE.MoscoviczF.MerelliA.LazarowskiA.et al (2021)

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Lecanemab in early Alzheimers disease

Previous study showed that MMP-2 and MMP-9 prompt CNV by degrading and remolding endothelial basement membrane and extracellular matrix [51]