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Upon activation of STAT3 by the Met receptor at the plasma membrane, it translocates to the nucleus to operate as a transcription factor regulating the expression of genes implicated in cell proliferation and differentiation (Y

Induction of epithelial tubules by growth factor HGF depends on the STAT pathway

Furthermore, the persistence of ALB expression despite declining hepatic protein levels supports the notion of functional heterogeneity, potentially reflecting a dynamic interplay between hepatocyte populations and expanding mesenchymal cells

The three components and their documented roles: Anchor-compound logic

Detailed understanding of the inhibitory action exerted by targeted RTK inhibitors including those affecting the HGF/c-Met signaling is critical for the durable antitumor responses