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Supplementation of cystine restored mTORC1 signaling and treatment with rapamycin (a specific mTORC1 inhibitor) abolished the induction, providing evidence for the direct role of mTORC1 in the phosphorylation of p70S6K (T389) and S6 (S235/236)

(2) Exogenous GSH effectively eliminated HT-induced oxidative damage not only by increasing activities of antioxidant enzymes but also by improving transcription regulation of the antioxidant responses

This substance is well known for its capacity to enhance skin suppleness, stimulate the production of collagen, and quicken the healing of wounds

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