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These regulatory systems enable cancer cells to enhance their adaptive capacity when confronted with oxidative stress damage induced by treatment, thereby playing a pivotal role in the formation of DTP

The pathological consequences of chronic inflammation in cancer are profound: persistent cytokine signaling enhances tumor cell proliferation and survival, while upregulation of immune checkpoint molecules like PD-L1 suppresses cytotoxic T cell responses, facilitating immune evasion [48]

Zarjou A, Bolisetty S, Joseph R, Traylor A, Apostolov EO, Arosio P, et al

Molybdenum and/or cadmium induce NLRP3 inflammasome production by causing mitochondria-associated endoplasmic reticulum membrane dysfunction in sheep hepatocytes

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