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Overexpression of glutaredoxin protects cardiomyocytes against nitric oxide-induced apoptosis with suppressing the S-nitrosylation of proteins and nuclear translocation of GAPDH

T1D is also characterized by immune dysregulation, accompanied by detectable autoantibodies

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Poster Session I - DIGITAL TRANSFORMATION, AI AND ROBOTICS 07.00 - DIGITAL TRANSFORMATION, AI AND ROBOTICS - 07.01 - TECHNOLOGY INNOVATIONS: ROBOTS, VIRTUAL REALITY, ARTIFICIAL INTELLIGENCE AND MORE P312 - ESOC25-859 DEEP-LEARNING TO IDENTIFY ARTEFACTUAL HYPODENSITY REGIONS IN RELATIVE NON-CONTRAST COMPUTED TOMOGRAPHY MAPS Praneeta R Konduri 1 , Lieselotte Vandewalle 1,2,3 , Soren Christensen 1 , Pierre Seners 4,5 , Anke Wouters 2 , Jelle Demeestere 2,3 , Robin Lemmens 2,3 , Maarten Lansberg 1 1 Department of Neurology and Neurological Sciences, Stanford University, Palo Alto, United States, 2 Department of Neurology, University Hospitals of Leuven, Leuven, Belgium, 3 Division of Experimental Neurology, Department of Neurosciences, KU Leuven, Leuven, Belgium, 4 U1266, INSERM, Institut de psychiatrie et Neurosciences de Paris (IPNP), Paris, France, 5 Department of Neurology, Hopital Fondation A

The result is a vicious cycle where declining NAD+ leads to worse metabolic function, which can lead to weight gain, which increases NNMT activity further