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The pathophysiology of DN is multifaceted, encompassing genetic susceptibility, perturbations in glucose metabolism, alterations in renal hemodynamics and the involvement of inflammatory mediators (2)

In fibroblasts derived from patients suffering from MPS types I, II, IIIA, and IIIB, genistein partially corrected the disturbances in the MPS II cell cycle and increased the tendency of MPS IIIA and IIIB fibroblasts to enter the S and G 2 /M phases (Moskot et al., 2016)