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These harmful variations disrupt the function of the glucocerebrosidase enzyme, leading to reduced enzymatic activity and potentially increasing the aggregation of -Syn (Sidransky et al

Feng F, Zhang J, Wang Z, Wu Q, Zhou X

It has been observed that the conversion from 2-hydroxysetrogens to 4-hydroxyestrogen might be an additional pro-inflammatory signal because these endogenous metabolites can be oxidized to 3,4-quinone, which lead to depurination and mutation [49]