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In lung metastases, tumor cells exploit pulmonary surfactant lipids, particularly phosphatidylcholines, to support membrane synthesis and evade oxidative stress, while alveolar macrophages shift toward an anti-inflammatory, lipid-laden phenotype

* Correspondence: Yunzhu Yang, [email protected] These authors have contributed equally to this work and share first authorship Disclaimer All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers

doi: 10.1016/j.jaci.2006.08.022

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Seizures and epilepsy after ischemic stroke