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doi:10.1007/s12178-025-09990-7 Chang CH, Tsai WC, Lin MS, Hsu YH, Pang JHS

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Many cardiovascular risk factors such as diabetes mellitus, hypertension and hyperlipidemia are demonstrated to stimulate the interaction of Cav-1 and eNOS, reduce nitric oxide production resulting in endothelial dysfunction and subsequently the formation of atherosclerotic lesion 41,42,43