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Furthermore, it has been shown that various antioxidant agents (eg, N -acetylcysteine and glutathione peroxidase) can substitute for Bcl-2 expression in preventing factor-deprived cells from undergoing apoptosis (Hockenbery et al, 1993)

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In this review, we aim to discuss lipid peroxidation-derived 4-HNE and MDA in the progression of OA

Glutathione in the treatment of liver diseases: insights from clinical practice states, Judging on the basis of the evidence obtained from experimental research and previous clinical studies, GSH administration seems to be a promising strategy to recover oxidative stress-induced liver damages in alcoholic and non-alcoholic liver diseases. Glutathione supplementation is a critical part of a two-pronged approach to help the liver stay healthy and combat alcohol damage, which includes: Lifestyle choices: Limit alcohol and other toxins that you can control, like tobacco, as well as environmental stressors like pesticides and excessive UV rays

However, also for GSTs, the mechanisms of protection are not always clear