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Feature · Product Review
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Description

While mutant mice deficient in Dvl1 or Dvl2 alone were normal, double knockouts of both Dvl1 and Dvl2 exhibit PCP defects (Wang et al., 2005), suggesting a functional redundancy between these 2 members

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Cell 61 (2016) 654666

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AudritoVManagoAZamporliniFRulliEGaudinoFMadonnaGet al

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The role of protein conjugation in the disposition of dinitrofluorobenzene

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