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Therefore, we determined whether TPH1 null mice had an altered metabolism of acetaminophen

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PGC-1 directs the exercise-related mitochondrial biogenesis, resulting in increase in oxygen supply to the muscle 3 and over-generation of ROS accompanied by electron leakage from the mitochondrial respiratory chain 4

The final efficacy was evaluated based on the RECIST 1.1 standard [145, 146] and confirmed clinical endpoints such as objective response rate and progression-free survival

Precursor ions were singly charged [M+H] for all analytes except GSSG, which showed both singly ([M+H], m/z = 613) and doubly ([M+2H] 2 , m/z = 307) charged ions