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Figure 2 visually integrates this framework, illustrating both the common pathways (e.g., mitochondrial dysfunction, gutliver axis disruption) and their disease-specific manifestations, while emphasizing the bidirectional interplay between liver pathology and magnesium homeostasis

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Acta Physiol Scand 161, 379384

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Using these for your clients is risky and can get you in real trouble

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Mitochondrial reactive oxygen species promote production of proinflammatory cytokines and are elevated in TNFR1-associated periodic syndrome (TRAPS)

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