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Immunologic deviations in recurrent spontaneous abortion mouse model

It is believed that S-glutathionylation is a regulatory mechanism that protects proteins from irreversible oxidation of sulfhydryl groups by oxidative stress to sulfinic and sulfonic acids resulting in protein degradation [155]

These in vivo systems capture complex immune responses and tissue-specific effects that are difficult to replicate in vitro, thereby enhancing the translational relevance of functional findings

Inhibition of histone deacetylase 1 or 2 reduces induced cytokine expression in microglia through a protein synthesis independent mechanism