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[DOI] [PubMed] [Google Scholar] 47.Li H.T., Jiao M., Chen J., Liang Y

We can see a large number of articles using glutamate modeling to study the mechanism of excitotoxicity, most of which only look at the increase in calcium ions and mitochondrial dysfunction caused by excitotoxicity

Near-infrared light-responsive, pramipexole-loaded biodegradable PLGA microspheres for therapeutic use in Parkinsons disease

In most cases, FAD is caused by mutations in two different genes: (i) The first type of mutations is localized in the genes encoding for the proteins presenilin 1 ( Psen1 ) and/or presenilin 2 ( Psen2 )

Our results are in line with these findings, where MSG resulted in impaired autophagy, verified by the prominent hampering in the gene expression of AMPK/SIRT-1/Beclin-1 pathway, together with upregulation in the gene expression of m-TOR, compared to the normal control group