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High maximal and spare respiratory capacity in seal cells after hypoxia exposure occurred in concert with increasing mitochondrial branch length and independent from major changes in extracellular acidification rate, suggesting seal cells recover oxidative metabolism without significant glycolytic dependency after hypoxia exposure

www.cdc.gov

In certain pathological conditions (such as periodontitis), high levels of methyl mercaptan (MeSH) act as a competitive false substrate for SQOR, reacting with the enzyme to form persulfide products (MeSSH) that cannot be further metabolized