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Rubino FM (2015) Toxicity of glutathione-binding metals: a review of targets and mechanisms

The mechanism involves -KG enabling the TCA cycle to operate fully, generating substantial amounts of NADH and FADH

We assessed the frequency of progenitor cells for MSCs (CFU-Fs) per 1x10e6 BM-MNCs as well per ml BM, MSC phenotype, proliferation capacity (doubling time-DT) as well as their differentiation potential in osteoblasts, adipocytes and chondrocytes

Sustained cysteine supply via SLC7A11 maintains high intracellular GSH levels, which are required to support the catalytic activity of downstream effectors, such as GPX4