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Increased IFN--producing Th17/Th1 cells and their association with lung function and current smoking status in patients with chronic obstructive pulmonary disease

CBRs may attenuate pro-inflammatory cytokine secretion by interfering with the phosphorylation of mitogen activated protein kinases (MAPK), such as extracellular signal-regulated kinase (ERK), which is known to participate in pro-inflammatory pathways, as shown in microglial cell line cultures (Eljaschewitsch et al., 2006