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Cisplatin-resistant OSCC cells suppress ferroptosis through upregulation of the SLC7A11-GSH-GPX4 axis, while reactivating ferroptosis can restore cisplatin sensitivity and reverse the resistant phenotype ( inhibitor erastin promotes ferroptosis via a ROS-dependent mechanism, enhances cisplatin cytotoxicity in OSCC cells, and exhibits no overt toxicity in vivo , providing a clinically translatable approach to overcome cisplatin resistance

Researches related to plant oxidative stresses have shown excellent potential for the development of stress-tolerant crops

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In an in vivo epilepsy model, 40 mg/kg and 60 mg/kg of GiGA1 exhibited antiseizure effects (Zhao et al., 2020) (Table 1)

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