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This new research provides a plausible biological explanation

The increase in nitric oxide is particularly intriguing since alpha-1 antitrypsin has been shown to inhibit iNOS, nitric oxide release and NF-kB activation, suggesting that alpha-1 antitrypsin replacement therapy or other neutrophil elastase inhibitor therapies may help decrease oxidative stress and inflammation

Exosomes generated through the endosomal pathway can be engineered for selective incorporation of small RNAs or proteins by exploiting ESCRT-dependent mechanisms, tetraspanin-associated sorting, or fusion tags such as Lamp2b and CD63 motifs (257)

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& Tanner, K