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Today, the global annual acetaminophen market is an astounding nine and a half billion dollars, fueled by heavy promotion that emphasizes the lack of gastrointestinal side effects associated with aspirin and other non-steroidal anti-inflammatory drugs (NSAIDS)

Given the progressive nature of the disorder, disease pathogenesis is mainly triggered by continual processes such as geometric accumulation of oxidative radicals brought about by cellular inclusions and mitochondrial dysfunction 8,9,10

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Stoyanov et al., 2011), with HIF-1 being a key molecule mediating this chondrogenic promotion effect (Wang W

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