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Ursolic Acid Induces Mitochondrial Biogenesis through the Activation of AMPK and PGC-1 in C2C12 Myotubes: a Possible Mechanism Underlying its Beneficial Effect on Exercise Endurance

Thus, TNBS promotes two independent mechanisms of injury, GSH depletion and ROS generation, both being required for the manifestation of mucosal injury as GSH limitation renders intestine susceptible to the TNBS-induced ROS overgeneration

This size-based penetration advantage is central to its topical research applications and helps explain why laboratory studies have consistently demonstrated biological activity following topical application models

Financial support No funding agency was involved in the writing of this review article

Every department has its own practices in place