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Observational human studies link higher TMAO levels with cardiovascular risk

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Paradoxically, while ABA promotes the expression of specific PYL isoforms (e.g., PYL7/9/11/12/13 ), it suppresses others (e.g., PYR1/PYL1/2/4/6/8 ) (Zhao et al, 2020), a functional dichotomy mirrored in GA signaling where GA represses GID1a/b/c transcription, whereas GA biosynthesis inhibitors (e.g., uniconazole) strongly induce their expression (Iuchi et al, 2007)