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Nonspecific disruption of copper homeostasis disrupts normal mitochondrial respiration, redox balance, and metalloenzyme function in healthy tissues, leading to organ damage

Dysfunctional AT leads to low adiponectin and high leptin levels, causing hepatic insulin resistance and increased lipolysis, further accelarating the development and progression of NAFLD

As an emerging metabolic-epigenetic regulatory mechanism, Kla plays a core role in reshaping cell fate by targeting ferroptosis in inflammatory injury, neurodegenerative diseases, cancer and I/R injury, altering disease outcomes through actions on key targets

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