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EGCG has been reported to exert its inhibitory effect on platelet viability through several mechanisms: the inhibition of collagen-mediated phospholipase (PL) Cgamma2, blockade of protein tyrosine phosphorylation, and the enhancement of Ca 2(+) -ATPase activity, thereby reducing platelet aggregation and alleviating atherothrombosis (140)

Your clinician decides whether you should stay at a dose, increase it, delay an increase, or make another adjustment based on your response, blood sugar trends, health history, and side effects