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The hexosamine pathway can be activated to enhance O-GlcNAc acylation, potentially leading to IR (Chen Y

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At centrosomes, glutamylation anchors the NEDD1 (Neural precursor cell-expressed, developmentally downregulated protein 1)/CEP192/-tubulin complex through electrostatic interactions (Fig

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Juckett G, Hartman-Adams H, Management of Keloids and Hypertrophic Scars, Am Fam Physician, 80(2009) pp.253-260

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Given the high heterogeneity, further studies are needed to confirm the results

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These findings are in line with the patterns that emerged upon observation of flux changes through central carbon metabolism pathways (Fig

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