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Whereas the the selective BET protein inhibitor apabetalone restored endothelial migration and tube formation in high-glucosetreated endothelial cells, suppressed the upregulation of the anti-angiogenic molecule thrombospondin-1 by preventing BRD4/H3K27ac-mediated chromatin activation and THBS1 transcription, preserved VEGFA signaling, and improved vascular perfusion and limb vascularization in diabetic models of endothelial dysfunction and hind limb ischemia [224]

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10.3389/fphys.2016.00234 122 LiuN.WangD.TianJ.WangX.ShiH.WangC.et al (2023)