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Thymosin beta-4 is a low-molecular-weight protein with a well-documented role in tissue repair processes, including wound healing, angiogenesis and peptide signaling, cellular proliferation and migration, re-epithelialization, and modulation of inflammatory responses

Inhibition of AMPK by LPS may therefore represent another means by which LPS promotes mTOR activity and consequently HIF-1 signaling, which could be another mechanism by which LPS induces the metabolic switch in innate immune cells

Standardized uptake value ratios were calculated using the cerebellum as the reference, with images obtained 85115 min for [18 F]Flutemetamol post-injection and 6090 min for [11 C]PIB

emission, 460 nm) were recorded over a period of 30 min using an Infinite M200 PRO multimode microplate reader (Tecan)