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The observation of intracellular accumulation of HNE-specific epitopes in human SLE for the first time raised the possibility that the higher lipid peroxidation may be directly involved in the pathogenesis of autoimmune disorders

Based on the outcomes of an investigation conducted by Zhou et al., EXOs derived from ovarian follicular fluid reduced PTEN transcription and lowered apoptosis

This damage can result in mutations within oncogenes such as RAS and MYC, as well as tumor suppressor genes like p53 and PTEN, thereby fostering genomic instability and malignant transformation [31, 32]

Sotalol and acebutolol can produce QTc prolongation due to potassium channel blockade, which may result in torsades de pointes (63)