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Intervention in the formation of PD is mainly carried out through editing of genes related to mitochondrial damage and autophagy, SNCA accumulation, and stabilization of oxidative phosphorylation (Figure 2)

Heart and Circulatory Physiology 243 (5): H691H697

The study revealed that viral infections or severe cellular distress trigger a state of Endoplasmic Reticulum (ER) stress, which induces the abnormal overproduction of the WASF3 protein

YTHDF1/GSTM2 axis sensitizes MRTK cells to ferroptosis in a GPX4-indpendent manner As shown above, YTHDF1 knockdown sensitized G401 cells to erastin-induced ferroptosis, and GSTM2 was identified as a critical target of YTHDF1